Test Code CTSU Ceramide Trihexosides and Sulfatides, Random, Urine
Necessary Information
Biochemical Genetics Patient Information (T602) is recommended. This information aids in providing a more thorough interpretation of results. Send information with specimen.
Specimen Required
Patient Preparation: Baby wipes or wipes containing soaps and lotions should not be used prior to urine collection because these may interfere with results.
Supplies: Sarstedt Aliquot Tube, 5 mL (T914)
Container/Tube: Plastic, 5-mL urine tube
Specimen Volume: 2 mL
Collection Instructions: Collect a first-morning, random urine specimen.
Specimen Stability Information: Refrigerated (preferred) 45 days/Ambient 45 days/Frozen 19 months
Forms
1. Biochemical Genetics Patient Information (T602)
2. If not ordering electronically, complete, print, and send a Biochemical Genetics Test Request (T798) with the specimen.
Useful For
Identifying patients with Fabry disease
Identifying patients with metachromatic leukodystrophy
Identifying patients with saposin B deficiency
Identifying patients with multiple sulfatase deficiency
Identifying patients with mucolipidosis II (I-cell disease)
Testing Algorithm
For information see:
-Fabry Disease Diagnostic Testing Algorithm
If the patient has abnormal newborn screening results for Fabry disease. Refer to the appropriate ACMG Newborn Screening ACT Sheet.(1)
Special Instructions
Method Name
Matrix-Assisted Laser Desorption/Ionization Time-of-Flight Mass Spectrometry (MALDI-TOF MS)
Reporting Name
Ceramide Trihex and Sulfatide, USpecimen Type
UrineSpecimen Minimum Volume
0.5 mL
Specimen Stability Information
Specimen Type | Temperature | Time | Special Container |
---|---|---|---|
Urine | Refrigerated (preferred) | 45 days | |
Ambient | 45 days | ||
Frozen |
Reference Values
An interpretive report will be provided.
Interpretation
The pattern of ceramide trihexosides or sulfatide excretion will be described. A normal pattern of excretion suggests absence of these diseases (see Cautions).
Evidence of ceramide trihexoside accumulation suggests decreased or deficient alpha-galactosidase activity, see Fabry Disease Testing Algorithm.
Evidence of sulfatide accumulation suggests decreased or deficient arylsulfatase A activity. Follow-up with the specific enzyme assay is recommended:
-ARSAW / Arylsulfatase A, Leukocytes
-ARSU / Arylsulfatase A, 24 Hour, Urine
To exclude multiple sulfatase deficiency (MSD), determination of iduronate-2-sulfatase activity is recommended.
-I2SWB / Iduronate-2-Sulfatase, Leukocytes
-I2SB / Iduronate-2-Sulfatase, Blood Spot
Evidence of both ceramide trihexoside and sulfatide accumulation suggests a diagnosis of mucolipidosis II (I-cell disease) or saposin B deficiency. Follow-up testing to rule-out I-cell disease may include molecular analysis of the GNPTAB gene or measurement of serum hydrolases (NAGS / Hexosaminidase A and Total Hexosaminidase, Serum).
Molecular genetic testing is required to confirm saposin B deficiency.
For more information see Lysosomal Storage Disorders Diagnostic Algorithm, Part 2.
Clinical Reference
1. ACMG Newborn Screening ACT Sheets. Accessed October 30, 2023. Available at www.acmg.net/ACMG/Medical-Genetics-Practice-Resources/ACT_Sheets_and_Algorithms/ACMG/Medical-Genetics-Practice-Resources/ACT_Sheets_and_Algorithms.aspx?hkey=9d6bce5a-182e-42a6-84a5-b2d88240c508
2. Desnick RJ, Ioannou YA, Eng CM. Alpha-galactosidase A deficiency: Fabry disease. In: Valle DL, Antonarakis S, Ballabio A, Beaudet AL, Mitchell GA. eds. The Online Metabolic and Molecular Bases of Inherited Disease. McGraw Hill; 2019. Accessed November 29, 2023. Available at https://ommbid.mhmedical.com/content.aspx?bookid=2709§ionid=225546984
3. Kuchar L, Ledvinova J, Hrebicek M, et al. Prosaposin deficiency and saposin B deficiency (activator-deficient metachromatic leukodystrophy): report on two patients detected by analysis of urinary sphingolipids and carrying novel PSAP gene mutations. Am J Med Genet A. 2009;149A(4):613-621
4. Mehta A, Hughes DA. Fabry disease. In: Adam MP, Feldman J, Mirzaa GM, et al, eds. GeneReviews [Internet]. University of Washington, Seattle; 2002. Updated March 9, 2023. Accessed November 29, 2023. Available at www.ncbi.nlm.nih.gov/books/NBK1292/
5. Schlotawa L, Ennemann EC, Radhakrishnan K, et al. SUMF1 mutations affecting stability and activity of formylglycine generating enzyme predict clinical outcome in multiple sulfatase deficiency. Eur J Hum Genet. 2011;19(3):253-261
6. Gieselmann V, Ingeborg K. Metachromatic leukodystrophy. In: Valle DL, Antonarakis S, Ballabio A, Beaudet AL, Mitchell GA. eds. The Online Metabolic and Molecular Bases of Inherited Disease. McGraw Hill; 2019. Accessed November 29, 2023. Available at https://ommbid.mhmedical.com/content.aspx?bookid=2709§ionid=225546629
7. Leroy JG, Cathey SS, Friez MJ. GNPTAB-related disorders. In: Adam MP, Feldman J, Mirzaa GM, et al, eds. GeneReviews [Internet]. University of Washington, Seattle; 2008. Updated August 29, 2019. Accessed November 29, 2023. Available at www.ncbi.nlm.nih.gov/books/NBK1828/
Day(s) Performed
Monday
Report Available
8 to 15 daysPerforming Laboratory
Mayo Clinic Laboratories in RochesterTest Classification
This test was developed and its performance characteristics determined by Mayo Clinic in a manner consistent with CLIA requirements. It has not been cleared or approved by the US Food and Drug Administration.CPT Code Information
83789
LOINC Code Information
Test ID | Test Order Name | Order LOINC Value |
---|---|---|
CTSU | Ceramide Trihex and Sulfatide, U | 59462-2 |
Result ID | Test Result Name | Result LOINC Value |
---|---|---|
606148 | Interpretation | 59462-2 |
606149 | Reviewed By | 18771-6 |